Deciphering the signaling network of breast cancer improves drug sensitivity prediction


Date

2021-05-19

Publication Type

Journal Article

ETH Bibliography

yes

Citations

Altmetric

Data

Abstract

One goal of precision medicine is to tailor effective treatments to patients’ specific molecular markers of disease. Here, we used mass cytometry to characterize the single-cell signaling landscapes of 62 breast cancer cell lines and five lines from healthy tissue. We quantified 34 markers in each cell line upon stimulation by the growth factor EGF in the presence or absence of five kinase inhibitors. These data—on more than 80 million single cells from 4,000 conditions—were used to fit mechanistic signaling network models that provide insight into how cancer cells process information. Our dynamic single-cell-based models accurately predicted drug sensitivity and identified genomic features associated with drug sensitivity, including a missense mutation in DDIT3 predictive of PI3K-inhibition sensitivity. We observed similar trends in genotype-drug sensitivity associations in patient-derived xenograft mouse models. This work provides proof of principle that patient-specific single-cell measurements and modeling could inform effective precision medicine strategies.

Publication status

published

Editor

Book title

Journal / series

Volume

12 (5)

Pages / Article No.

401 - 418000000000000

Publisher

Cell Press

Event

Edition / version

Methods

Software

Geographic location

Date collected

Date created

Subject

cell lines; breast cancer; single-cell signaling; drug sensitivity prediction; mechanistic modeling; Cellular signaling; proteomics; EGF-MAP kinase pathway

Organisational unit

03927 - Picotti, Paola / Picotti, Paola check_circle
09735 - Bodenmiller, Bernd / Bodenmiller, Bernd check_circle

Notes

Funding

866004 - Three-dimensional dynamic views of proteomes as a novel readout for physiolgical and pathological alterations (EC)
823839 - European Proteomics Infrastructure Consortium providing Access (EC)

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