Translation is required for miRNA‐dependent decay of endogenous transcripts


Loading...

Date

2021-02-01

Publication Type

Journal Article

ETH Bibliography

yes

Citations

Altmetric

Data

Abstract

Post-transcriptional repression of gene expression by miRNAs occurs through transcript destabilization or translation inhibition. mRNA decay is known to account for most miRNA-dependent repression. However, because transcript decay occurs co-translationally, whether target translation is a requirement for miRNA-dependent transcript destabilization remains unknown. To decouple these two molecular processes, we used cytosolic long noncoding RNAs (lncRNAs) as models for endogenous transcripts that are not translated. We show that, despite interacting with the miRNA-loaded RNA-induced silencing complex, the steady-state abundance and decay rates of these transcripts are minimally affected by miRNA loss. To further validate the apparent requirement of translation for miRNA-dependent decay, we fused two lncRNA candidates to the 3'-end of a protein-coding gene reporter and found this results in their miRNA-dependent destabilization. Further analysis revealed that the few natural lncRNAs whose levels are regulated by miRNAs in mESCs tend to associate with translating ribosomes, and possibly represent misannotated micropeptides, further substantiating the necessity of target translation for miRNA-dependent transcript decay. In summary, our analyses suggest that translation is required for miRNA-dependent transcript destabilization, and demonstrate that the levels of coding and noncoding transcripts are differently affected by miRNAs.

Publication status

published

Editor

Book title

Volume

40 (3)

Pages / Article No.

Publisher

EMBO Press

Event

Edition / version

Methods

Software

Geographic location

Date collected

Date created

Subject

Dicer knockout mESC; long noncoding RNAs; miRNA; RNA metabolic labelling; translation

Organisational unit

03983 - Ciaudo, C. (ehemalig) / Ciaudo, C. (former) check_circle

Notes

Funding

173120 - Canonical and non-canonical functions of RNA interference proteins during mouse early development (SNF)

Related publications and datasets