Antibiotic-recalcitrant Salmonella exploits post-antibiotic microbiota disruption to achieve virulence-dependent transmission


Date

2025-07-22

Publication Type

Journal Article

ETH Bibliography

yes

Citations

Altmetric

Data

Abstract

Antibiotic-recalcitrant reservoirs contribute to clearance failure and relapse of bacterial infections in vivo. Salmonella Typhimurium survives antibiotic exposure in host tissue, later resuming replication and reseeding the gut lumen. It is not well understood how relapsing infection is shaped by Salmonella virulence factors nor how antibiotic therapy affects transmission from infected animals to new hosts. Here, we study how antibiotic therapy affects Salmonella during long-term systemic infection of 129/SvEv mice. Plateable pathogen loads decline during ceftriaxone treatment but gradually increase after the end of antibiotic therapy, and this regrowth is promoted by intracellular virulence factors. We observe massive pathogen blooms in the gut, which lead to clonal transmission and invasive infection of cagemates. Transmission is blocked when treated animals are co-housed with naive cagemates that harbor intact microbiota reservoirs, which confer community colonization resistance. Our work provides new insights into how antibiotic-recalcitrant tissue reservoirs are shaped by pathogen-host-microbiota dynamics.

Publication status

published

Editor

Book title

Journal / series

Volume

44 (7)

Pages / Article No.

115969

Publisher

Cell Press

Event

Edition / version

Methods

Software

Geographic location

Date collected

Date created

Subject

antibiotic persistence; recalcitrance; persisters; Salmonella; host-pathogen interactions; transmission; animal models

Organisational unit

03589 - Hardt, Wolf-Dietrich / Hardt, Wolf-Dietrich check_circle

Notes

Funding

173338 - Deciphering the initial steps that lead to Salmonella Typhimurium diarrhea (SNF)
192567 - Mechanisms controlling the Salmonella Typhimurium gut infection (SNF)

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