Preservation of cell-survival mechanisms by the presenilin-1 K239N mutation may cause its milder clinical phenotype
- Journal Article
Journal / seriesNeurobiology of Aging
Pages / Article No.
SubjectAkt; Amyloid-β; Apoptosis; MAP kinase signaling; Oxidative stress; Presenilin 1 mutation
NotesReceived 22 May 2016, Revised 4 July 2016, Accepted 4 July 2016, Published online 15 July 2016.
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