Preservation of cell-survival mechanisms by the presenilin-1 K239N mutation may cause its milder clinical phenotype
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Date
2016-10Type
- Journal Article
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Cited null times in
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Publication status
publishedExternal links
Journal / series
Neurobiology of AgingVolume
Pages / Article No.
Publisher
ElsevierSubject
Akt; Amyloid-β; Apoptosis; MAP kinase signaling; Oxidative stress; Presenilin 1 mutationNotes
Received 22 May 2016, Revised 4 July 2016, Accepted 4 July 2016, Published online 15 July 2016.More
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Citations
Cited null times in
Web of Science
Cited 3 times in
Scopus
ETH Bibliography
yes
Altmetrics