A spontaneous leptin receptor point mutation causes obesity and differentially affects leptin signaling in hypothalamic nuclei resulting in metabolic dysfunctions distinct from db/db mice
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https://doi.org/10.3929/ethz-b-000351256Publication status
publishedExternal links
Journal / series
Molecular MetabolismVolume
Pages / Article No.
Publisher
ElsevierSubject
Leptin receptor mutation; Ventromedial nucleus; Dorsomedial nucleus; Arcuate nucleus; Energy expenditure; Glucose toleranceOrganisational unit
03596 - Kopf, Manfred / Kopf, Manfred
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