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dc.contributor.author
Naef, Roland
dc.contributor.supervisor
Eppenberger, Hans Max
dc.contributor.supervisor
Suter, Ueli
dc.date.accessioned
2017-06-13T02:47:01Z
dc.date.available
2017-06-13T02:47:01Z
dc.date.issued
2000
dc.identifier.uri
http://hdl.handle.net/20.500.11850/144551
dc.identifier.doi
10.3929/ethz-a-003885341
dc.format
application/pdf
dc.language.iso
en
dc.publisher
ETH Zürich
dc.rights.uri
http://rightsstatements.org/page/InC-NC/1.0/
dc.subject
MYELINPROTEINE
dc.subject
GENMUTATIONEN + PUNKTMUTATIONEN (GENETIK)
dc.subject
NEUROPLEXUS + PERIPHERES NERVENSYSTEM (NEUROPATHOLOGIE)
dc.subject
NERVLICHE UND PSYCHIATRISCHE ERBKRANKHEITEN
dc.subject
SCHWANN'SCHE ZELLEN (CYTOLOGIE, HISTOLOGIE)
dc.subject
MYELIN PROTEINS
dc.subject
GENE MUTATIONS + POINT MUTATIONS (GENETICS)
dc.subject
NEUROPLEXUS + PERIHERAL NERVOUS SYSTEM (NEUROPATHOLOGY)
dc.subject
HEREDITARY NERVOUS AND PSYCHIATRIC DISORDERS
dc.subject
SCHWANN CELLS (CYTOLOGY, HISTOLOGY)
dc.title
A common disease mechanism for hereditary neuropathies due to point mutations in the peripheral myelin protein 22
dc.type
Doctoral Thesis
dc.rights.license
In Copyright - Non-Commercial Use Permitted
ethz.size
93 Bl.
ethz.code.ddc
DDC - DDC::6 - Technology, medicine and applied sciences::610 - Medical sciences, medicine
ethz.notes
Diss. Naturwissenschaften ETH Zürich, Nr. 13530, 2000.
ethz.identifier.diss
13530
ethz.identifier.nebis
003885341
ethz.publication.place
Zürich
ethz.publication.status
published
ethz.leitzahl
ETH Zürich::00002 - ETH Zürich::00012 - Lehre und Forschung::00007 - Departemente::02030 - Dep. Biologie / Dep. of Biology::02539 - Institut für Molecular Health Sciences / Institute of Molecular Health Sciences::03367 - Suter, Ulrich / Suter, Ulrich
ethz.date.deposited
2017-06-13T02:47:19Z
ethz.source
ECOL
ethz.identifier.importid
imp59366a2d6971d27203
ethz.ecolpid
eth:23419
ethz.eth
yes
ethz.availability
Open access
ethz.rosetta.installDate
2017-08-01T17:57:57Z
ethz.rosetta.lastUpdated
2020-02-15T00:28:32Z
ethz.rosetta.versionExported
true
ethz.COinS
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